Cerebral Atrophy Essay

598 words - 3 pages

Cerebral Atrophy
Blood vessels carry oxygen and nutrients to the brain. When these blood vessels are blocked by a clot strokes occur.
Symptoms of cerebral atrophy include dementia, seizures, loss of motor control, and difficulty with speaking, comprehension or reading
Diagnosing cerebral atrophy can be achieved by use of a CT scan , MRI scan , PET scan , or a Single-photon emission computed tomography (SPECT).
Because there is no cure for cerebral atrophy, treating the symptoms become more of the focus. Treatment includes medications, physical therapy and psychological counseling and support.
If no treatment occurs then a person may not be able to participate in normal activities, lose their independence or become depressed.

Cardiac Hypertrophy
When there is increased stress on the heart, hypertrophy of the heart muscle occurs. There are two ventricles of the heart; the right ventricle ...view middle of the document...

Echocardiogram and EKGs are also used in diagnosing cardiac hypertrophy.
Depending on the underlying cause of the condition will determine whether surgery or medication is needed. Medications include: Angiotensin-converting enzyme (ACE) inhibitors, Angiotensin II receptor blockers (ARBs), Thiazide diuretics Beta blockers and/or Calcium channel blockers
If cardiac hypertrophy is not treated heart failure, arrhythmia, ischemic heart disease, heart attack or cardiac arrest may occur.

Summary Post
As a result of this week’s discussion, I’ve learned more about the changes in cells and tissues after an injury has occurred. There were a few conditions that could be treated with medication or surgery, while others have no cure at all. I got a better understanding of cerebral atrophy and cardiac hypertrophy. At first, I couldn’t quite grasp the pathophysiology of dysplasia as the result of an abnormal pap smear but after reading a few DQ post, I’m a bit more informed than when I first read about it in my textbook. The preventive measures for cervical dysplasia are practiced through making positive lifestyle choices. Overall, pathophysiology seems as if it’s going to be an interesting topic to learn.

Lab
Summary Post
This week, we touched on the metric system. We learned the basic units of measure and the different methods for conversion. I’ve relearned facts about the metric system and dosage calculations in adults and children. I’m glad there was a math review portion in our reading. It’s amazing what we forget, when it’s not practiced on a regular basis. I picked back up on it pretty quickly though. I’ve also learned why the metric system is only used in US hospitals.

When blood flows too slowly, cells in the blood begin to stick together and form clumps. High counts of red blood cells and particles, low temperatures, and high concentrations of fat can lead to an increase in blood thickness. When blood flows too fast, this means there is more fluid in the blood. This causes low blood pressure.
Smith, S. (2012). What is blood viscosity? Retrieved from http://www.wisegeek.com/what-is-blood-viscosity.htm

Other Papers Like Cerebral Atrophy

Central Nervous System Essay

3858 words - 16 pages PCB 3702 Chapter 8 The Central Nervous System Learning Objectives 1. Describe the embryonic origin of the CNS 2. Identify the 5 brain regions & the major structures they contain, including the ventricles 3. Describe the organization of the sensory & motor areas of the cerebral cortex, & the nature of the basal ganglia 4. Distinguish between the functions of the right & left cerebral hemispheres, and describe the significance of

Alzheimer Disease Essay

922 words - 4 pages The documentation of a severe form of dementia by Alois Alzheimer in 1907 began a massive investigation of the cause of this disorder. Some of the common symptoms of Alzheimer's Disease consist of memory loss, impaired language ability, impaired judgement, and learning (M. Wong, et al. , 1997). Alzheimer's Disease (AD) is mainly a disease of the cerebral cortex. Alzheimer's is marked structurally by the senile plaques, neurofibrillary tangles

Late Adulthood

991 words - 4 pages of their physical strength and endurance during their twenties and then gradually decline, in later adulthood, a variety of physiological changes start to occur, including some degree of atrophy of the brain and a decrease in the rate of neural process. The respiratory and circulatory system are less efficient, bone mass diminishes, especially in women, leading to osteoporosis. The muscles become weaker unless exercise programs are followed. The

Managing Client with Cerebrovascular Disease

3878 words - 16 pages the skull to allow the brain to swell. In the patient’s case, the intra-operation report indicated that upon successful exposure, the brain was “angry-looking” as quoted in the report. A photograph of the brain was taken for educational purposes and I can recall that the vessels were very thick and protruding out. A large subdural haematoma was evidently seen overlying the cerebral hemisphere and as planned, evacuation of the haematoma was

Notes for 3rd Year

2205 words - 9 pages hypercoagubility o hyperlipidaemia o obesity o Oral contraceptive use o Physical inactivity o Sickle cell disease Inferior view of cerebral circulation: Blood flow ▪ The brain requires a continuous supply of blood to provide the brain with oxygen and glucose ▪ Blood flow must be maintained at 750- 1000mls/ minute Interrupted blood flow ▪ If interrupted (eg cardic arrest) ▪ Neurological metabolism is

The Genetics of Huntingtons Disease

1805 words - 8 pages characterized by slow gradual personality changes, dementia, and chorea form movements. It is a progressive disease; its average onset is thirty to forty years of age, and the duration of the disease is about ten to twenty years with death as the outcome. It is known that for Huntington’s disease, there is a degeneration of cholinergic and GABAergic neurons in the basal ganglia and the cerebral cortex. (Ball, Harper 1992) The etiology of nerve

Vision Impairment in Older Adults

1530 words - 7 pages prevent ischemia and necrosis) may also limit the extent of the trauma and save some functions (surgery performed on B.L. relieving pressure and stabilizing the fracture). Immediately after the injury it is difficult to adequately assess the extent of the injury. This is because of the conduction of impulses ceases in the cerebral cortex and in the nerve fibers at the side of injury and slightly above it - a situation known in medical world as a

Alzheimer

4642 words - 19 pages having an aggressive form of dementia, manifesting in memory, language and behavioral deficits3. Dr. Alzheimer noted many abnormal symptoms, including difficulty with speech, agitation, and confusion4. He followed her care for five years, until her death in 1906. Following her death, Dr. Alzheimer performed an autopsy, during which he found dramatic shrinkage of the cerebral cortex, fatty deposits in blood vessels, and atrophied brain cells2. He

Almost All Causes of Coma

5280 words - 22 pages GLUTEN SENSITIVE ENTROPATHY.... remember this by BROW B=Barley R=Rye O=Oat W=Wheat Cerebellar arteries occlusion SCA--->AHDH MCA (AICA)--->AHDH+5,7,8 ICA (PICA)--->AHBH+V+9,10 Superior cerebral artery occllusion A Ataxia H Horner's D Deafness H Contralateral hemihyposthesia (Face+Body ipsilateral) Middle Cerebral Artery Occlusion (AICA) A Ataxia H Horner's D Deafness H Contralateral hemihyposthesia (Face ipsilateral+Body contralateral

Brain of Einstein

2962 words - 12 pages % formalin by injection into the internal carotid arteries was carried out, and the whole brain was then freely suspended in 10% formalin for fixation and subsequent study. No significant neuropathology was seen on examination (gross or microscopic). After fixation, caliper measurements were made directly from the brain; calibrated photographs were taken of all views of the whole brain and of the dissected hemispheres; the cerebral hemispheres were cut

Diencephalic Syndrome: a Case Report

3964 words - 16 pages suprasellar mass. A chronic subdural hematoma was also noted on the left cerebral convexity, which is a complication of the VP shunt. Obstructive hydrocephalus was also noted to be persistent (Figure 6).Due to the signs of increasing intracranial pressure and the increase in size and extent of the suprasellar mass, surgical intervention was suggested. Even if she was nutritionally impaired, she underwent right pterional, right frontoparietal craniotomy

Related Essays

Multiple Sclerosis: Changes Beyond Demyelination Essay

1573 words - 7 pages ). Gray matter T2 hypointensity is related to plaques and atrophy of multiple sclerosis patients. Journal of Neurological Sciences, 185, 19-26. Bear, M. F., Connors, B. W., & Paradiso, M. A. (2001). Neuroscience: Exploring the brain, 2nd ed. Baltimore, MD: Lippincott, Williams & Wilkins. Evangelou, N., Konz, D., Esiri, M. M., Palace, J., & Matthews, P. M. (2000). Regional axonal loss in the corpus callosum correlates with cerebral white matter

Spinocerebellar Ataxia Essay

921 words - 4 pages Spinocerebellar Ataxia (SCA) Last updated November 2010 Spinocerebellar ataxia (SCA) is one of a group of genetic disorders characterized by slowly progressive in-coordination of gait and often associated with poor coordination of hands, speech, and eye movements. Frequently, atrophy of the cerebellum occurs. As with other forms of ataxia, SCA results in unsteady and clumsy motion of the body due to a failure of the fine coordination of

Topical Morphine Essay

1038 words - 5 pages include cerebral atrophy, brainstem lesions, and temporal lobe disease. Table 1 Conditions Associated with Musical Hallucinations Hypacusis Psychiatric Disorders Depression Schizophrenia Obsessive-compulsive disorders Structural Brain Lesions Cerebral atrophy Temporal lobe lesions Epilepsy Brainstem lesions Brain tumors Cerebral vascular accident/multi-infarct dementia Drugs Ketamine Ranitidine Prazosin Bromocriptine

Dementia Essay

607 words - 3 pages independently can decline, leaving thembed/chair bound;The person's capacity for self-care progressively diminishes, making them totallydependent on carers;The person's ability to eat independently gradually disappears, often in associationwith a diminished ability to swallow and increased risk of aspiration. A progressiveloss of appetite almost always follows; andOther complications can include bowel and bladder incontinence, muscle atrophy